HomeresearchRoundworm larvae in the blood - more common than expected

Roundworm larvae in the blood – more common than expected

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Parasites are repeatedly found in our patients, with possible implications for multiple diseases

Dr. Simon Yu – the only true expert

Parasites have been a regular feature in our practice since our course with Dr. Simon Yu in St. Louis, the world’s most experienced parasite expert. Unfortunately, darkfield blood testing is so time-consuming that we don’t perform it routinely, because surprisingly, we repeatedly find clear evidence of parasites when we examine the blood in this way.

Simon treats patients with multiple medications simultaneously, and for a long time. If the liver is “damaged,” he says, “you have to give more medication.” Our experience confirms this: the treatments are extremely well-tolerated!

His latest book, by the way, is extraordinary and should be highly recommended to every holistic practitioner. It was just published. I met Simon at my lecture to the Biological Dentists in London and received a first copy! Thank you, Simon!

 

COPD patient yesterday

A patient came in with “chronic obstructive pulmonary disease” that suddenly appeared shortly after a vacation in Thailand, essentially the “asthma of aging” or of chronic smokers. He smoked 30 cigarettes a day until he became ill. He lived in a moldy house until he became ill.

Because we learned, especially from Klinghardt, that many COPD diseases are caused by parasites, I examined his blood and promptly found a roundworm larva.

Here is the corresponding image; serology is still pending.

V. Ascaris findings after frequent Thailand vacation

Thailand often problematic

Further down, I write about a patient in a coma with cerebral parasitosis after a Thailand vacation. Two weeks ago, I had a patient who, after an animal bite in Thailand, had a chronic sand flea infection. The flea larvae he pulled out of his skin and brought back with him look like… It looks almost like Morgellons, and the historical picture of Morgellons may actually involve sand flea larvae:

 

Similar: Dominican Republic

Darkfield of Blood of my sister Ilse – she was 12 years earlier in Dominican Republic in Holiday and was severely sick afterward. Obviously strongyloides larvae infestation in her blood.

 

 

 

 

case of massivest roundworm infestation

In 2017, we had a patient with Ascaris, which was actually serologically confirmed at the Drop Institute, who then excreted large clumps of worms with our enemas. Ascaris (roundworm) grows up to 30 cm long.

After several weeks of excreting up to 10 fist-sized clumps of worms a day with our enemas – see photos below – as well as several weeks of albendazole therapy, I still found an Ascaris larva in the darkfield blood.

(The diagram of the parasite has been copied into the image; you can clearly see that the rear part of the larva is swimming in the blood):

 

Here are the excreted worms in the toilet.

(c) Dr. Helmut Retzek, Ascaris suis in a patient’s stool – discharge through special enemas

Fist-sized worm clumps

12This patient with the large numbers of worms was bedridden for seven years. During several hospital stays, nothing was found, and he was classified as “mentally ill.” A two-year course of antibiotic therapy for Borrelia had little effect.

Video about a severe roundworm infestation

https://www.youtube.com/watch?v=hyYduvsQ60M

 

Unclear suspicion of roundworm in a patient

Here is another patient with suspected Ascaris infestation in the dark field. The clinical symptoms could be consistent (rheumatic pain, periodic worsening), unclear serological findings (“slightly positive”)

 

Most extreme: Ascaris brain infection in one of our patients

Highly interesting case from 2019: Patient begins experiencing epileptic seizures after a vacation in Thailand, initially only at night, later also during the day. A lumbar puncture at Vienna General Hospital revealed ASCARIS antibodies, and treatment consists of antiepileptic drugs. He progressively loses consciousness and eventually falls into a coma.

Anti-Lyme hyperthermia in Bad Aibling brings 10% improvement; rudimentary contact (via the eyes) is possible.

Dr. Simon Yu measures him in our office in spring 2019 using AMA and puts together a five-way combination of various anti-parasitic medications. With each cycle of treatment, extreme deterioration (epileptic seizures, psychosis, hallucinations, etc.) in the sense of a Herxheimer reaction occurred briefly, but overall, the recovery was miraculous. Even the Vienna General Hospital supported the treatment and also prescribed rituximab (antibodies against TNF-alpha to minimize encephalitis).

 

Conclusion: Parasitoses: Difficult Diagnosis – Difficult Therapy

Serological diagnostics are difficult; we only very rarely see clean and proper antibodies in patients, whom we very clearly diagnose for parasites bioenergetically according to Simon Yu using AMA or our RAC/polarizing filter test.

Eosinophils are usually absent anyway (in an Australian study, only 11% of affected parasite patients had elevated Eosinophils).

This weakness of serology is well known in the scientific literature; I suspect it occurs similarly to chronic neuroborreliosis: some patients can be misled by the parasitic organisms, which produce TGF-ß, which shuts down the immune system.

Possibly. Is it a genetic predisposition or a history of chronic persistent EBV or CMV infection? These viruses are known to reduce the nonspecific immune system to 3% (see my lecture on VDR blockade, where the studies are located).

The dark-field microscope is most likely to be helpful for diagnosis. I’ve never seen stool samples positive for parasite eggs in my life.

 

For example, a filiary/nematode larval infestation in a Parkinson’s patient yesterday.

Dead filiary larva: the intestine is already distended, typical shape and covered with multiple leukocytes (as evidence of endogenous origin, no contamination), approximately 15 µm thick, several hundred µm long.

(c) Dr. Retzek, July 2019 – Filiarien larva in darkfield blood of a Parkinson’s patient with coronary heart disease

 

Worm egg in dark field examination

A filiary worm egg in the blood is known to have a diameter of 20-60µm, i.e., 3-10 erythrocyte widths. Incidentally, this came from the dark field blood of my then chronically ill daughter, who had a CMV titer >10,000 and was thus practically immune-compromised. Since a parasite treatment, she’s been doing great.

Parasite egg in the blood of Dr.’s daughter. Retzek (c) Retzek 2018
Oxyura larva in my daughter’s blood – the darkfield scan is performed by Nadine Bukovsky “MediMikro”

 

And here is the corresponding Wikipedia image of an Oxyura head (pinworm, Enterobios vermicularis, threadworm).

By the way, we had NEVER seen worms in her stool!

 

(c) Wikipedia

 

Could this be a lint?

Are what we find artifacts? This is what a lint looks like in darkfield:

Clearly outside the plane of the erythrocytes (here with a 4x objective), clear, flat structure, no intestine, no pointed or typical ends, not covered by granulocytes.

(c) Dr. Retzek July 2019 – Lint on the coverslip in the dark field

Are autoimmune diseases of parasitic origin?

Dr. Alan Macdonald, emeritus pathologist in the USA, has been able to detect parasites in the brain in all MS (multiple sclerosis) cases examined.

Alan Macdonald: Multiple Sclerosis is a parasitosis

Therefore, it would also be logical that with conventional immunosuppressive therapy for multiple sclerosis, so many patients fall into the secondary chronic progressive form if the actual cause of an autoimmune disease were an infection.

Studies show IL-17 production by parasites

Autoimmunity arises from TH-17 (mis-)programmed lymphocytes, which occurs through the expression of interleukin 17.

Parasites also produce their their own IL-17, which immunologically paralyzes the person; the affected person attacks themselves (autoimmune disease) and no longer the parasites that colonize and exploit them.

Interleukin-17 then produces the autoimmune cells TH-17, which trigger autoimmune diseases!

 

EBV & CMV could be door openers

Epstein-Barr virus, CMV, and other herpes viruses contribute to this (–>studies), chronic EBV persistence despite supposedly curative antibodies (“EBNA”) is virtually unknown in conventional medicine.

We have observed persistence or reactivation in practice in patients with an EBV virus titer of > 200, purely clinically. From 1000 IU, the patients are ill, and from 5000 IU, they are usually unable to work.

The concept of “Non-healing of an infection despite antibodies” when considering HIV (AIDS):

Antibodies are present here too, yet the virus persists and kills those infected.

  • EBV survives in the B cells and glands (thyroid, liver, spleen).
  • HIV in the T cells.

Persistent EBV or CMV (or HZV or HSV) –> according to our laboratory measurements, is usually a contributing factor to autoimmune diseases, cancer, and burnout.

EBV is promoted by electrosmog (Marshall 2017, Maisch 1998, Feychting 2005, Klinghardt – numerous lectures –> hence the sharp increase in these diseases today.

I will continue to report, Dr. Retzek July 2019

this posting was never published, only 2025 I found it and put it public

 

Update 2025

  • all parasite infected patients have heavy-metals and almost all have KPU.
  • If our “liver-cleansing” protocol does not work within one month, then parasites are present

 

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