HomeresearchAlzheimer's Dementia - Update 2024

Alzheimer’s Dementia – Update 2024

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The background for this Alzheimer’s article 2024 is my research on Parkinson’s 2023, where I was able to unravel the essential neuro-apoptotic mechanisms and their treatment.

 

In short – the Parkinson’s mechanism

  • oxidative stress caused by melamine deposits x metals in the cells – leads to oxidative damage of structural proteins – in Parkinson’s it is SYNUCLEIN.
  • the synuclein precipitates in the cell
  • this “dirt” triggers an “unfolding protein system” to initiate APOPTOSIS –> Cell dead
  • alternative trigger mechanisms for Parkinson’s are: Nocardia infection (like tuberculosis), probably also Lyme disease and other infections, increased cytokine triggering by RANTES from the hidden dental foci, magnesium deficiency (extremely strong pro-inflammatory stimulus for the brain).

 

I have found answers for each of these steps

basically this is the blueprint for all neurodegenerative diseases

 

Alzheimer

In Alzheimer’s disease, it is the amyloid deposits of phosphorylated TAU protein as well as intercellular ß-amyloids.

  • Here we already know that the ß-amyloids are actually the body’s own antibiotics, especially against e.g. spirochetes (Lyme disease)  and   fungi.
  • We know that we can successfully dissolve the TAU deposits with SPERMIDIN (wheat germ), thanks to the research of the well-known Austrian doctor Prof. Reinhart Jarrisch, which is also well published.
  • In my own practice, I have brought mild Alzheimer’s patients back with Trazodone
  • We have also experienced success with the help of biological Borrelia remedies (wormwood wine) in cases of early dementia, which we were able to significantly improve by administering targeted antibiotics.
  • Virus infection – often persistent in the brain – is known to cause problems there: EBV and herpes (eg MS, Alzheimer’s), also COVID (Alzheimer’s), Borna (depression) – can be treated not so badly with medication (Valacyclovir, Tenoforvir, Ivermectin) and phytos (Cistus …) and also OZONE.
  • Aluminium is now ubiquitous as an additive thanks to “solar radiation management” and is known to cause Alzheimer’s disease (50,000 scholar entries), which can be yes.constantly eliminate.
  • Stimulation of the glymphatic system in the brain through LFMS and the brain stem stimulator (after all 40,000 scholar entries) is certainly of great importance – because it costs practically nothing and, based on the studies, brings clear results – much better than the super-expensive antibodies – something like this will never find its way into conventional medicine.
  • Transcranial brain stimulation as we learned it from Petros is probably the best therapy for these patients, as Petros demonstrates again and again, we have a lot of patients on our homepage as testimonial videos.
  • Dr. Nehls is currently traveling around the world preaching LITHIUM against Alzheimer’s, has written books and articles on the background and treatment of Alzheimer’s
Summary of the article by Dr. Nehls

The most important statements in the article on the Unified Theory of Alzheimer’s Disease (UTAD) by Michael Nehls are:

  1. AD as a result of lifestyle and environmental factors:
    • Alzheimer’s is caused primarily by lifestyle and environmental factors, not by aging alone.
    • An unhealthy western lifestyle (e.g. lack of exercise, poor diet, chronic stress) impairs neurogenesis and promotes AD.
  2. Role of adult hippocampal neurogenesis (AHN):
    • AHN, the formation of new nerve cells in the hippocampus, is crucial for memory and stress resilience.
    • Factors such as exercise, social interaction and intermittent fasting promote AHN.
    • Disturbances of AHN lead to cortisol overproduction, depression and an increased risk of AD.
  3. “Law of the Minimum” (LOM):
    • AHN is limited by the scarcest essential element. Deficits in one area (e.g. exercise, diet) cannot be compensated by others.
    • Prevention and treatment of AD require holistic approaches that improve multiple lifestyle factors simultaneously.
  4. Amyloid-beta and tau:
    • Amyloid-beta has physiological functions in memory formation; however, its excess leads to toxic oligomers.
    • Tau pathology is a major factor in the progression of AD.
  5. Evolutionary perspective:
    • Alzheimer’s is rare under “natural” conditions (e.g. pre-industrial lifestyle).
    • The article argues for considering AD as a disease caused by lifestyle deficiencies.
  6. Therapeutic approaches:
    • A systems biology, personalized approach could prevent or cure AD in the early stages.
    • Effective prevention includes regular exercise, social connections, nutrient-rich diet and intermittent fasting.

The focus of the article is a holistic view of Alzheimer’s pathogenesis with a focus on modifiable lifestyle factors.

Dr. In his article, Nehls recommends the following nutritional factors for the prevention and possible treatment of Alzheimer’s:

  1. Mediterranean diet (MeDi):
    • Rich in olive oil, legumes, whole grains, fruits, nuts and vegetables.
    • Moderate to high fish consumption, reduced meat intake.
    • Proven positive effects on brain structures and less brain atrophy.
  2. N-3 fatty acids (DHA and EPA):
    • Essential for neuroprotective and anti-inflammatory functions.
    • Promotes adult hippocampal neurogenesis (AHN).
  3. Vitamins:
    • B vitamins (especially B6, B9, B12): Reduction of homocysteine, a risk factor for cognitive disorders.
    • Vitamin D: Essential for neuroprotection and cognitive functions, often underdosed in prevention.
  4. Polyphenols and antioxidants:
    • Resveratrol (in grapes): Reduces oxidative stress and amyloidogenic processes.
    • Foods rich in polyphenols (e.g. berries): Support the AHN.
  5. Selenium and zinc:
    • Necessary for antioxidant enzyme functions and neurogenesis.
  6. Intermittent fasting (IMF):
    • 12+ hours of nightly fasting promotes ketogenesis and brain metabolism.
  7. Avoiding risk factors:
    • Reducing meat consumption, which promotes oxidative stress and inflammatory processes.
    • Limiting AGEs (Advanced Glycation End Products) that are produced by highly processed foods

 

Dr. Nehls recommends substituting the following micronutrients in the prevention and treatment of Alzheimer’s

 

  1. Vitamin B complex (B6, B9, B12):
    • Reason: Reduction of homocysteine, which has a neurotoxic effect and inhibits neurogenesis.
    • Dosage: Adjustment depending on homocysteine ​​level, e.g. high-dose combination preparations in the case of elevated homocysteine.
  2. Vitamin D:
    • Reason: Protection against cognitive decline and promotion of neurogenesis.
    • Dosage: Serum target values ​​of 50–70 nmol/L; typically 2000–4000 IU/day.
  3. Selenium:
    • Rationale: Reduction of oxidative stress that affects AHN.
    • Dosage: Target values ​​around 85 μg/L serum.
  4. Zinc:
    • Rationale: Essential for neurogenesis and degradation of amyloid-beta.
    • Dosage: Depends on measured serum value.
  5. N-3 fatty acids (DHA, EPA):
    • Rationale: Neuroprotective effects and anti-inflammatory.
    • Dosage: 1–2 g DHA/EPA per day.
  6. Alpha-lipoic acid (ALA):
    • Rationale: Antioxidant and promotes mitochondrial function.
    • Dosage: 600 mg/day.
  7. Lithium (low dose):
    • Rationale: Promotes AHN and reduces GSK-3β activity, which drives Alzheimer’s pathogenesis.
    • Dosage: 300 µg/day in drinking water.
  8. Pyrroloquinoline quinone (PQQ):
    • Reason: Promoting mitochondrial function.
    • Dosage: 20 mg/day.

All micronutrients should be individually adjusted and taken under medical supervision

 

Based on the above summary and the research that has been going on for many, many years, we do the following for Alzheimer’s:

1) Diagnostics

  • Analysis of the infectious pathogens (Borrelia, Babesia, Bartonella, Rikettsia, Aspergillus, Candida, various herpes, EBV, CMV)
  • Analysis of the dental foci (with an overview X-ray) –> If the patient is not interested in eliminating NICO and root canal treatments, we will stop the treatment, as we have more patients than places and we need to use our resources optimally.
  • Urine analysis by Tobias
  • Stool analysis  microbiome
  • Metal analysis: I don’t believe in that anymore, I think the laboratories were forced to keep the values ​​low. 20 years ago we usually had 10-100 times the load in the DMPS excretion tests.
  • our bioenergetic testing

 

2) Therapy

  • Immediate start with brain stimulation (tDCS) and vagus stimulator (taVNS), I also strongly recommend buying the LFMS.
  • Micronutrients: Lithium, MultiB, Benfothiamine, Omega3, VitC/D/E, Q10, Boron
  • Change diet to 16/8 – low-carb or even ketogenic – according to Nehls
  • Administration of C11 – caprylic acid in food
  • “Brain demucus with Wormwood
  • Trittico (Trazodone) as a UPS blocker
  • Haritaki – from Ayurveda
  • Spermidine as an autophagy inducer to reduce tau amyloids
  • Sports program – exercise is a must for rebuilding memory
  • Bifidus – intestinal structure
  • Aluminum elimination, heavy metals

 

3) specific infection therapy

if one of the co-triggering infections is clearly evident in the laboratory and bioenergeticallyis ethically positive, appropriate anti-infective therapy – we now prefer to simply use medication and antibiotics

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